BLOOD CLOTS as soon as you eat this food - What are they hiding from people? / Fizuli Huseynov
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#BirBax #FizuliHuseynov #pressure Phytotherapist Dr. Fizuli Huseynov Herba Medical Center: (+994) 55 304 26 79 #халгтабабети абабати #сагламлік #thromb #blood #heart functioning in a healthy person, the platelet-vascular system and coagulation hemostasis ensure the completeness of the vessel wall. Normally, when small vessels are damaged, bleeding stops in 1-2 minutes. This is mainly due to the deposition and aggregation of platelets, and to a lesser extent due to spasm of vessels. The leading role in this process is played by damage to the blood vessel wall, grafting of the subendothelial tissue structure, especially collagen. Rapid activation of platelets occurs under the influence of Willebrand factor contained in collagen and subendothelium. At this time, platelets change their shape, swell, form a thorn-like protrusion, adhere to the connective tissue fiber at the edge of the wound. The deposition of platelets on the subendothelium of the damaged vessel wall is considered the initial stage of vascular thrombohemostasis. At the same time, the aggressiveness of catecholamines, serotonin and callogenic platelets released from damaged cells increases. At this time, platelet-containing substances with electron density are formed from platelets. These substances increase blood clotting ability. It should be remembered that platelets affect the intensity and speed of local coagulation in the thrombus-forming area. Blood clotting occurs as a result of the interaction of platelet factors released from plasma and tissue platelets. A small amount of thrombin is generated in the area of primary hemostasis. On the one hand, this completes the irreversible process of platelets, and on the other hand, it causes the formation of fibrin. Activation of platelet-vascular hemostasis in patients with coronary atherosclerosis takes an active part in the process of developing coronary heart failure. Multiple defects of the endothelial cover are one of the causes of thrombus formation. Thus, the formation of thrombus in the blood of people with ischemic heart disease, as well as the fragment broken off from the thrombus, lead to the formation of embolism. This condition is most common in patients with unstable angina and acute heart attack. Risk factors for thrombus formation: ischemic heart diseases, hyperlipidemia, diabetes mellitus, high arterial pressure, thrombogenic factors, smoking, negative emotions, obesity, sedentary lifestyle, hereditary factors, disorders of the blood coagulation system. Blood thrombus formation can cause the following diseases: angina pectoris, heart rhythm disorders, heart failure
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